10–14 Nov 2025
Office of Grants and Research
Africa/Accra timezone

Targeting Bacterial Virulence: Salacia debilis Phytochemicals Disrupt Quorum Sensing and Efflux Mechanisms in Pseudomonas aeruginosa and Staphylococcus aureus

13 Nov 2025, 13:30
15m
Office of Grants and Research

Office of Grants and Research

Oral Presentation Health Systems, Basic sciences, Biomedical Advances, pharmaceutical Sciences and Human Wellbeing

Speaker

Ms Philipina Acheampong (KNUST, department of chemistry)

Description

Philipina Acheampong¹, Michael Konney Laryea¹, and Lawrence Sheringham Borquaye¹,²
¹Department of Chemistry, Kwame Nkrumah University of Science and Technology (KNUST), Ghana
²Central Laboratory, Kwame Nkrumah University of Science and Technology (KNUST), Ghana
Email: philipinacheampong32@gmail.com

Antimicrobial resistance (AMR) remains a critical global health challenge, largely driven by multidrug-resistant ESKAPE pathogens (E. faecium, S. aureus, K. pneumoniae, A. baumannii, P. aeruginosa, and Enterobacter spp.). In Ghana, AMR-related infections accounted for an estimated 4.95 million deaths in 2019. With decreasing efficacy of current antibiotics, antivirulence strategies such as quorum sensing (QS) and efflux pump inhibition offer promising alternatives that reduce pathogenicity without exerting strong selective pressure. Salacia debilis, a medicinal shrub from the Celastraceae family, is traditionally used to treat malaria. SD-03 (Benzyl 2-methoxybenzoate), SD-04 (1,10-dihydroxy-6H-benzo[c]chromen-6-one), and SD-05 (2,4-dimethoxy-8-methyldibenzo[b,d]furan-1-carboxylic acid) previously isolated from S. debilis, have shown moderate antimalarial and antimicrobial activity. However, there is no information on their QS and efflux pump inhibitory activities. This study, thus investigates the QS and efflux pump inhibitory activities of S. debilis extracts and compounds, using molecular docking and molecular dynamics simulations to identify potential protein targets. Crude and ethyl acetate extracts inhibited the growth of S. aureus and P. aeruginosa with minimum inhibitory concentrations ranging from 6.25 to 25.00 mg/ml. Molecular docking revealed strong interactions between SD-03, SD-04, and SD-05 with key QS proteins (LasR, RhlR, PqsR) and the NorA efflux pump protein. LasR binding affinities were -9.8, -10.4, and -6.6 kcal/mol, respectively. These preliminary results suggest that S. debilis compounds may have antivirulence potential by targeting QS and efflux mechanisms. Further in vitro and in silico studies are needed to confirm these effects and explore their mechanisms of action.
Keywords: Salacia debilis; antimicrobial resistance; ESKAPE pathogens; antivirulence; quorum sensing; efflux pump; molecular docking.

Primary authors

Prof. Lawrence Sheringham Borquaye (KNUST, department of chemistry) Dr Michael Konney Laryea (KNUST, department of chemistry) Ms Philipina Acheampong (KNUST, department of chemistry)

Co-author

Ms Khyrat Yahaya (KNUST, department of chemistry)

Presentation materials

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